Topic/Source: The Blood Clotting Cascade. Vessel injury triggers two pathways. Extrinsic Pathway (FAST): Tissue Factor activates Factor VII. Intrinsic Pathway (SLOW): Exposed collagen activates Factor XII. Both merge at Factor X. Factor X converts Prothrombin into Thrombin. Thrombin converts Fibrinogen into Fibrin. Fibrin mesh traps RBCs to form a clot. Style: Serious Subject. Design: Clean white background, dark navy text, teal accents. Minimalist medical aesthetic. No stock photos of random people.
Topic/Source: The Blood Clotting Cascade. Vessel injury triggers two pathways. Extrinsic Pathway (FAST): Tissue Factor activates Factor VII. Intrinsic Pathway (SLOW): Exposed collagen activates Factor XII. Both merge at Factor X. Factor X converts Prothrombin into Thrombin. Thrombin converts Fibrinogen into Fibrin. Fibrin mesh traps RBCs to form a clot. Style: Serious Subject. Design: Clean white background, dark navy text, teal accents. Minimalist medical aesthetic. No stock photos of random people.
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The coagulation cascade begins with vessel damage, triggering both the extrinsic and intrinsic pathways. The extrinsic pathway is rapid, activated by tissue factor, while the intrinsic pathway is slower, initiated by collagen. Both pathways converge at Factor X, which transforms prothrombin into thrombin. Thrombin then converts fibrinogen into fibrin, forming a mesh that traps red blood cells, effectively sealing the injury and restoring haemostasis.