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This presentation explores the complexities of high-flow arteriovenous malformations (AVMs) and their impact on cardiac load. We will define critical clinical questions and present the haemodynamic–microcirculatory hypothesis, alongside the authors' affiliations. The haemodynamic architecture of fast-flow AVMs will be examined, highlighting the bypassing of capillaries and the implications of increased venous pressure. We will discuss the pathobiology of these lesions, focusing on their...